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Time Facts, New York, NY (2026)

09/07/2026

CREATINE MAY DO MORE THAN SUPPORT MUSCLE — RESEARCH IS EXPLORING ITS ROLE IN IMMUNE FUNCTION AND CANCER

Creatine is best known for helping muscles produce energy.

But immune cells also need energy to function.

CREATINE → PHOSPHOCREATINE → ATP PRODUCTION → CELLULAR ENERGY

Research suggests creatine can influence immune-cell activity, including macrophages and other components of the immune response.

In a 2023 laboratory and mouse study, creatine supplementation increased ATP production in macrophages and enhanced their anti-tumor activity. The researchers also observed increased tumor-specific CD8+ T-cell responses and slower tumor growth in mice.

THE PROPOSED PATHWAY:

Creatine
→ more available cellular energy
→ enhanced macrophage activity
→ stronger tumor-antigen presentation
→ increased CD8+ T-cell response
→ greater anti-tumor activity in the experimental model

BUT THERE’S AN IMPORTANT CATCH:

The cancer-fighting findings are primarily from laboratory and animal research. We do not yet have enough human evidence to say that creatine prevents or treats cancer.

Even its effects on inflammation in humans remain uncertain. A 2026 systematic review of randomized trials found no significant overall reduction in inflammatory markers such as CRP or IL-6 with creatine supplementation.

So for now:

CREATINE = WELL-STUDIED FOR MUSCLE PERFORMANCE
IMMUNE FUNCTION = PROMISING AREA OF RESEARCH
CANCER TREATMENT = NOT PROVEN IN HUMANS

REFERENCES:
• “The Role of Creatine in the Development and Activation of Immune Responses” — Nutrients.
• “Creatine supplementation enhances anti-tumor immunity by promoting adenosine triphosphate production in macrophages” — Frontiers in Immunology.
• “Impact of creatine supplementation on inflammation: evidence from a systematic review and meta-analysis of randomized double-blind placebo trials” — Frontiers in Immunology, 2026.

DISCLAIMER:
This content is for educational and informational purposes only. It is not intended to diagnose, treat, cure, or prevent any disease or medical condition. Creatine should not be considered a cancer treatment or substitute for prescribed medical care. Laboratory and animal findings do not necessarily translate into benefits in humans. If you have a medical condition, take medications, or are undergoing cancer treatment, consult a qualified healthcare professional before using supplements.

09/07/2026

6-GINGEROL FROM GINGER MAY MAKE SOME CHEMOTHERAPY DRUGS MORE EFFECTIVE — IN LAB STUDIES

6-GINGEROL, ONE OF THE MAJOR BIOACTIVE COMPOUNDS IN GINGER, IS BEING STUDIED FOR ITS POTENTIAL TO WORK TOGETHER WITH CERTAIN CANCER DRUGS.

In laboratory studies:

6-GINGEROL + CHEMOTHERAPY
→ increased cancer-cell damage
→ enhanced apoptosis
→ disrupted cancer-cell survival pathways
→ potentially allowed lower drug concentrations to produce similar effects

In one breast cancer cell study, researchers found that 10 µM 6-gingerol could reduce the amount of paclitaxel needed by up to 4× to achieve a similar effect.

Another laboratory study found that 6-gingerol combined with 5-FU or paclitaxel produced substantially greater inhibition of cervical cancer cells than 6-gingerol alone.

6-GINGEROL MAY WORK BY:
6-Gingerol
→ disrupts cancer-cell signaling
→ affects cell-cycle control
→ activates apoptosis pathways
→ makes cancer cells more vulnerable to certain drugs

IMPORTANT: These are laboratory findings, not proof that eating ginger makes chemotherapy 4× or 8× more effective in cancer patients.

The concentrations of purified 6-gingerol used in cell experiments cannot simply be compared with the amount obtained from eating or drinking ginger.

INSTEAD, ENJOY GINGER AS PART OF A BALANCED DIET.

Fresh ginger can add flavor to tea, soups, stir-fries, marinades, and other foods. But ginger should never replace prescribed cancer treatment.

REFERENCES:
• “Anticancer Efficacy of 6-Gingerol with Paclitaxel against Wild Type of Human Breast Adenocarcinoma” — Molecules.
• “Assessment of anti-cancerous potential of 6-gingerol and its synergy with drugs on human cervical adenocarcinoma cells” — Food and Chemical Toxicology.
• “Protective and therapeutic potential of ginger extract and [6]-gingerol in cancer” — Phytotherapy Research.

DISCLAIMER:
This content is for educational and informational purposes only. It is not intended to diagnose, treat, cure, or prevent any disease or medical condition. Ginger and 6-gingerol should not be considered a cancer treatment or substitute for prescribed medical care. Laboratory findings do not necessarily translate into benefits in humans. Never start, stop, or change cancer treatment based on information from social media. Always consult a qualified healthcare professional regarding cancer treatment.

09/07/2026

BLACK PEPPER’S PIPERINE IS BEING STUDIED FOR ITS ANTI-CANCER POTENTIAL

Piperine, the compound responsible for black pepper’s spicy kick, has shown promising effects against prostate and colon cancer cells in laboratory studies.

Researchers have found that piperine may interfere with cancer-cell growth and survival and has also been investigated for its potential to help overcome drug resistance—a major challenge in cancer treatment.

But there’s an important distinction: these findings are mainly from laboratory and preclinical research. They do not prove that eating black pepper can prevent, treat, or cure cancer in people.

Black pepper remains a flavorful source of piperine, while scientists continue studying how this compound might be used in future cancer therapies.

09/07/2026

CURCUMIN FROM TURMERIC HAS SHOWN P53-DEPENDENT CANCER CELL DEATH IN BREAST CANCER LAB STUDIES

A COMPOUND FOUND IN TURMERIC IS BEING INVESTIGATED FOR HOW IT MAY DISRUPT THE SURVIVAL OF CERTAIN BREAST CANCER CELLS.

Curcumin is the major bioactive compound studied in turmeric.

In laboratory experiments using MCF-7 breast cancer cells, curcumin increased p53 activity and promoted expression of downstream proteins involved in programmed cell death, including Bax. Researchers found evidence that the effect was dependent on the p53 pathway.

HERE’S THE MECHANISM RESEARCHERS ARE INVESTIGATING:

CURCUMIN
↓
ACTIVATES p53
↓
INCREASES PRO-DEATH SIGNALING
↓
ACTIVATES APOPTOSIS
↓
BREAST CANCER CELL DEATH

Other laboratory studies have found that curcumin can also cause G2/M cell-cycle arrest in breast cancer cell lines while increasing p21 and altering proteins involved in cell-cycle control.

SPECIALIZED CURCUMIN FORMULATIONS ARE ALSO BEING STUDIED.

A laboratory study of curcumin and cyclodextrin formulations reported an IC50 of approximately 7.33 µg/mL for curcumin against MCF-7 cells. Cyclodextrin formulations are being investigated because ordinary curcumin has poor water solubility and limited bioavailability.

ANOTHER INTERESTING APPROACH IS NC2603.

NC2603 is a curcumin analog, meaning it is chemically related to curcumin but is not simply turmeric or ordinary dietary curcumin.

In MCF-7 cells, NC2603 showed an IC50 of 5.6 µM and altered genes involved in cell-cycle regulation, including increased CDKN1A/p21 signaling. Researchers also compared its effects with doxorubicin using MCF-7 and non-malignant HaCaT cells.

BUT HERE’S THE MOST IMPORTANT PART:

THESE ARE LABORATORY FINDINGS — NOT PROOF THAT EATING TURMERIC TREATS BREAST CANCER.

The experiments used purified compounds, specialized formulations, or synthetic analogs at controlled concentrations.

Those concentrations cannot simply be compared with the amount of curcumin obtained from using turmeric as a kitchen spice.

Curcumin also has poor bioavailability, which is one reason researchers are developing formulations and analogs designed to improve delivery. (PubMed)

INSTEAD, ADD TURMERIC TO YOUR DIET

Turmeric can be used as a flavorful spice in:

• Curries
• Soups
• Rice dishes
• Roasted vegetables
• Eggs
• Smoothies or golden milk

BOTTOM LINE:

CURCUMIN IS A PROMISING SUBJECT OF BREAST CANCER RESEARCH.

Laboratory studies suggest it can activate p53-dependent pathways, alter cell-cycle regulation, and promote programmed cell death in certain breast cancer cells.

But lab activity is not the same as a proven cancer treatment in humans.

REFERENCES:

• “Curcumin induces apoptosis in human breast cancer cells through p53-dependent Bax induction” — FEBS Letters (PubMed)
• “Curcumin inhibits proliferation and promotes apoptosis of breast cancer cells” — Oncology Reports (PubMed)
• “Design, Preparation and Evaluation of Supramolecular Complexes with Curcumin for Enhanced Cytotoxicity in Breast Cancer Cell Lines” — Pharmaceutics (PubMed Central (PMC))
• “Exploring CDKN1A Upregulation Mechanisms: Insights into Cell Cycle Arrest Induced by NC2603 Curcumin Analog in MCF-7 Breast Cancer Cells” — International Journal of Molecular Sciences (PubMed Central (PMC))

DISCLAIMER:
This content is for educational and informational purposes only. It is not intended to diagnose, treat, cure, or prevent cancer or any other disease. Turmeric and curcumin should not be considered cancer treatments or substitutes for prescribed medical care. Laboratory findings do not necessarily translate into benefits in humans. Specialized curcumin formulations and synthetic analogs used in research are not equivalent to ordinary culinary turmeric. Never start, stop, or change cancer treatment based on information from social media. Cancer treatment decisions should always be made with qualified healthcare professionals.

09/06/2026

CINNAMON TEA MAY SUPPORT HEALTHY BLOOD SUGAR — BUT IT IS NOT A PROVEN TREATMENT

CINNAMON CONTAINS COMPOUNDS THAT ARE BEING STUDIED FOR THEIR POTENTIAL EFFECTS ON GLUCOSE CONTROL.

CINNAMON
↓
BIOACTIVE COMPOUNDS
↓
MAY INFLUENCE INSULIN SIGNALING AND GLUCOSE METABOLISM
↓
MAY HELP SUPPORT BLOOD SUGAR CONTROL

Several clinical studies have investigated cinnamon supplementation in people with type 2 diabetes. A 2024 meta-analysis of 24 randomized controlled trials found that cinnamon supplementation was associated with improvements in fasting blood glucose, insulin resistance, and HbA1c.

The amount of active compounds extracted into a cup of cinnamon tea can vary considerably.

INSTEAD, ENJOY CINNAMON TEA AS PART OF A HEALTHY ROUTINE

You can make unsweetened cinnamon tea by steeping a cinnamon stick in hot water.

Avoid loading it with sugar or sweetened syrups, especially if you’re trying to manage blood glucose.

ONE MORE SAFETY NOTE:

Cassia cinnamon, the most common type sold in the U.S., contains coumarin, which can be harmful to the liver when consumed in large amounts over long periods. Ceylon cinnamon contains much less coumarin.

BOTTOM LINE:

CINNAMON MAY HAVE A MODEST ROLE IN SUPPORTING BLOOD SUGAR CONTROL, BUT CINNAMON TEA IS NOT A CURE FOR HIGH BLOOD SUGAR OR DIABETES.

If you have diabetes or take glucose-lowering medication, don’t replace prescribed treatment with cinnamon or herbal teas.

REFERENCES:

• “The effect of cinnamon supplementation on glycemic control in patients with type 2 diabetes mellitus: An updated systematic review and dose-response meta-analysis of randomized controlled trials” — Phytotherapy Research
• “A Systematic Review Evaluating Cinnamon’s Effects on Glucose Utilizing a Ranking System to Assess Bias and Study Quality” — Journal of Medicinal Food
• National Center for Complementary and Integrative Health — “Cinnamon: Usefulness and Safety”
• National Center for Complementary and Integrative Health — “Diabetes and Dietary Supplements: What You Need To Know”

DISCLAIMER:
This content is for educational and informational purposes only. It is not intended to diagnose, treat, cure, or prevent diabetes or any other medical condition. Cinnamon tea should not replace prescribed diabetes medication, a medically recommended diet, blood-glucose monitoring, or professional medical care. If you have high blood sugar or diabetes, consult a qualified healthcare professional.

09/06/2026

FDA APPROVES A NEW TARGETED THERAPY FOR ADVANCED PANCREATIC CANCER

A NEW DRUG IS GIVING PATIENTS WITH METASTATIC PANCREATIC CANCER ANOTHER TREATMENT OPTION

The U.S. FDA has approved Rasonque (daraxonrasib), a first-in-class targeted therapy for adults with metastatic pancreatic adenocarcinoma who have received at least one prior systemic therapy or are not candidates for multiagent systemic therapy.

HOW DOES IT WORK?

PANCREATIC CANCER CELL
↓
ABNORMAL RAS SIGNALING
↓
PROMOTES TUMOR GROWTH AND SURVIVAL
↓
DARAXONRASIB TARGETS RAS
↓
BLOCKS KEY CANCER-GROWTH SIGNALS
↓
HELPS SLOW DISEASE PROGRESSION

In the randomized clinical trial involving 500 adults with previously treated metastatic pancreatic adenocarcinoma, patients receiving Rasonque had:

MEDIAN OVERALL SURVIVAL
Rasonque → 13.2 months
Standard chemotherapy → 6.7 months

The FDA also reported improvements in progression-free survival and overall response rate compared with standard chemotherapy.

WHY THIS MATTERS:

Pancreatic adenocarcinoma is one of the most difficult cancers to treat, and RAS signaling has long been recognized as an important driver of pancreatic tumor growth.

Daraxonrasib represents a new approach: targeting RAS directly rather than relying only on conventional chemotherapy.

BUT THIS IS NOT A CURE.

The approval applies to a specific group of patients with metastatic pancreatic adenocarcinoma. Like other cancer medicines, Rasonque can cause side effects, including rash, diarrhea, mouth inflammation, nausea, fatigue, vomiting, abdominal pain, swelling, decreased appetite, and hemorrhage.

BOTTOM LINE:

A NEW TARGETED THERAPY HAS BEEN FDA-APPROVED FOR METASTATIC PANCREATIC CANCER.

Rasonque (daraxonrasib) is designed to attack RAS-driven cancer signaling and, in a randomized trial, extended median overall survival compared with standard chemotherapy.

It doesn’t mean pancreatic cancer has been cured — but it represents a significant new treatment option for eligible patients.

REFERENCES:

• “FDA Approves First in Class Targeted Therapy for Metastatic Pancreatic Cancer” — U.S. Food and Drug Administration, August 26, 2026.
• “FDA Permits Expanded Access for Investigational Pancreatic Cancer Drug” — U.S. Food and Drug Administration, May 1, 2026.

DISCLAIMER:
This content is for educational and informational purposes only. It is not intended to diagnose, treat, cure, or prevent cancer or any other medical condition. Rasonque (daraxonrasib) is approved only for specific patients and treatment circumstances. Treatment decisions should always be made by qualified oncology professionals based on the patient’s diagnosis, cancer type, stage, molecular characteristics, previous treatments, and overall health. Never start, stop, or change cancer treatment based on information from social media.

09/06/2026

CUCUMBER + PINEAPPLE CAN SUPPORT HYDRATION AND DIGESTIVE HEALTH — BUT IT DOESN’T “DETOX” YOUR COLON

THIS REFRESHING COMBINATION PROVIDES WATER, VITAMIN C, AND PLANT COMPOUNDS THAT CAN FIT INTO A HEALTHY DIET.

CUCUMBER
↓
HIGH WATER CONTENT
↓
HELPS SUPPORT HYDRATION

PINEAPPLE
↓
VITAMIN C + NATURAL CARBOHYDRATES
↓
PROVIDES NUTRIENTS AND ENERGY

IF YOU BLEND THE WHOLE FRUITS AND VEGETABLES
↓
YOU RETAIN MORE OF THEIR NATURAL FIBER
↓
FIBER HELPS SUPPORT NORMAL BOWEL MOVEMENTS
↓
SUPPORTS REGULAR DIGESTION

The digestive system already has its own waste-removal system. Your liver, kidneys, gastrointestinal tract, and other organs continuously process and eliminate waste, so special “detox” drinks are not necessary to cleanse the colon.

AND THERE’S AN IMPORTANT DIFFERENCE:

BLENDING ≠ JUICING

Blending whole cucumber and pineapple generally retains their fiber, while straining the mixture into juice removes much of that fiber.

Pineapple also contains bromelain, a group of enzymes that has been studied for various biological effects. However, drinking pineapple juice has not been proven to “clean out” the intestines or remove accumulated waste.

INSTEAD, TRY:

CUCUMBER + PINEAPPLE + WATER
→ BLEND WITHOUT STRAINING
→ ENJOY AS PART OF A BALANCED DIET

You can also add mint, lime, or plain yogurt for flavor and additional nutrients.

BOTTOM LINE:

CUCUMBER + PINEAPPLE CAN BE A HYDRATING, NUTRITIOUS BLEND — BUT IT IS NOT A COLON DETOX.

Keeping enough fiber, fluids, fruits, vegetables, and whole grains in your diet is a more evidence-based way to support regular bowel function.

REFERENCES:
• National Institute of Diabetes and Digestive and Kidney Diseases — Constipation and healthy bowel habits
• USDA FoodData Central — Cucumber and pineapple nutrient information
• National Center for Complementary and Integrative Health — Detoxes and cleanses

DISCLAIMER:
This content is for educational and informational purposes only. It is not intended to diagnose, treat, cure, or prevent any disease or medical condition. Cucumber and pineapple can be part of a healthy diet, but they should not be considered a colon cleanse, detoxification treatment, or substitute for medical care. If you have persistent constipation, abdominal pain, blood in your stool, or other digestive symptoms, consult a qualified healthcare professional.

09/06/2026

CLOVE CONTAINS EUGENOL, WHICH HAS SHOWN ANTIPARASITIC ACTIVITY IN LABORATORY STUDIES

A NATURAL COMPOUND FOUND IN CLOVE IS BEING STUDIED FOR ITS POTENTIAL EFFECTS AGAINST CERTAIN PARASITES.

Cloves are rich in eugenol, a bioactive compound that has demonstrated antimicrobial and antiparasitic activity in laboratory research.

HERE’S WHAT RESEARCHERS ARE INVESTIGATING:

CLOVE
↓
EUGENOL
↓
INTERACTS WITH PARASITE CELLS
↓
MAY DISRUPT CELLULAR FUNCTIONS
↓
INHIBITS OR DAMAGES CERTAIN PARASITES
↓
ANTIPARASITIC ACTIVITY

Laboratory studies have found that eugenol can affect certain parasitic organisms, including some protozoan and helminth species. These findings make eugenol an interesting subject for further antiparasitic research.

BUT THERE’S AN IMPORTANT LIMIT:

THIS IS LABORATORY RESEARCH

Studies often use purified eugenol at controlled concentrations that may be very different from the amount obtained by drinking clove tea or eating cloves.

INSTEAD, ENJOY CLOVE AS PART OF A BALANCED DIET

Cloves can add flavor to:
• Tea
• Oatmeal
• Curries
• Soups
• Baked foods

BOTTOM LINE:

EUGENOL FROM CLOVE HAS SHOWN ANTIPARASITIC ACTIVITY IN LABORATORY STUDIES, BUT HUMAN CLINICAL EVIDENCE IS STILL LIMITED.

Cloves should not replace proper testing or medically recommended treatment for a confirmed parasitic infection.

REFERENCES:
• Research on eugenol and antiparasitic activity in parasite models
• National Center for Complementary and Integrative Health — Herbal Medicines and Supplements
• Centers for Disease Control and Prevention — Parasites

DISCLAIMER:
This content is for educational and informational purposes only. It is not intended to diagnose, treat, cure, or prevent any disease or infection. Cloves and eugenol should not be considered a proven treatment for parasitic infections. If you suspect a parasite infection, consult a qualified healthcare professional for proper testing and treatment.

09/06/2026

CANCER VACCINES ARE BEING TESTED AGAINST ONE OF THE MOST AGGRESSIVE BRAIN CANCERS

RESEARCHERS ARE EXPLORING WHETHER A PERSONALIZED VACCINE CAN TRAIN THE IMMUNE SYSTEM TO RECOGNIZE AND ATTACK GLIOBLASTOMA CELLS

Glioblastoma is an aggressive primary brain cancer that remains difficult to treat.

Now, researchers are testing a different strategy: therapeutic cancer vaccines designed to help the immune system recognize features that are specific to a patient’s tumor.

HERE’S HOW A PERSONALIZED VACCINE CAN WORK:

TUMOR SAMPLE
↓
GENETIC SEQUENCING
↓
IDENTIFY TUMOR-SPECIFIC MUTATIONS
↓
SELECT NEOANTIGENS
↓
CREATE A PERSONALIZED VACCINE
↓
ACTIVATE T CELLS
↓
T CELLS RECOGNIZE TARGETS ON TUMOR CELLS
↓
IMMUNE SYSTEM ATTACKS THE CANCER

In a 2026 Phase 1 trial, researchers tested a personalized DNA vaccine targeting up to 40 tumor-specific neoantigens in patients with MGMT-unmethylated glioblastoma. The vaccine activated and expanded circulating T cells in nearly all evaluable patients, and no serious vaccine-related safety signals or dose-limiting toxicities were reported.

Other clinical trials are also testing different glioblastoma vaccine approaches, including personalized neoantigen vaccines, RNA-based vaccines and vaccines targeting proteins found in tumor cells.

THE BIG IDEA:

Instead of simply attacking cancer directly, researchers are trying to teach the immune system what the tumor looks like — potentially giving T cells a more precise target.

If successful, personalized cancer vaccines could become part of a future treatment strategy for glioblastoma.

BOTTOM LINE:

CANCER VACCINES ARE SHOWING PROMISING EARLY IMMUNE RESPONSES IN GLIOBLASTOMA RESEARCH.

But promising immune activity is not the same as a proven treatment. Clinical trials are still determining whether these vaccines can translate into longer survival and better outcomes for patients.

REFERENCES:

• “Adjuvant personalized multivalent neoantigen DNA vaccination for MGMT unmethylated glioblastoma: a phase 1 trial” — Nature Cancer
• “Immunological outcomes of a bespoke DNA cancer vaccine in glioblastoma” — Nature Cancer
• National Cancer Institute — Personalized DNA Vaccine for the Treatment of Newly Diagnosed Glioblastoma
• National Cancer Institute — RNA-Lipid Particle Vaccines for the Treatment of Patients with Recurrent Glioblastoma

DISCLAIMER:
This content is for educational and informational purposes only. It is not intended to diagnose, treat, cure, or prevent cancer or any other medical condition. Cancer vaccines for glioblastoma remain investigational and should not be considered a proven cancer treatment or substitute for established medical care. Early clinical-trial results do not establish effectiveness. Cancer treatment decisions should always be made with qualified healthcare professionals. Never start, stop, or change cancer treatment based on information from social media.

09/06/2026

DANDELION ROOT EXTRACT SHOWED OVER 95% INHIBITION OF COLON CANCER CELLS WITHIN 48 HOURS IN A LAB STUDY

A PLANT EXTRACT SHOWED STRIKING EFFECTS AGAINST COLON CANCER CELLS IN LABORATORY RESEARCH.

Researchers tested an aqueous dandelion root extract against human colorectal cancer cell lines, including HT-29 and HCT116.

DANDELION ROOT EXTRACT
↓
ACTIVATED MULTIPLE CELL-DEATH PATHWAYS
↓
DISRUPTED CANCER CELL SURVIVAL
↓
TRIGGERED PROGRAMMED CELL DEATH
↓
MORE THAN 95% OF THE TESTED CANCER CELLS WERE AFFECTED WITHIN 48 HOURS

The researchers also tested the extract in mouse models carrying human colorectal tumors. Tumor growth was significantly reduced, showing that the extract deserved further investigation. However, these results were preclinical and do not establish that dandelion root treats cancer in humans.

IMPORTANT:

The study used a concentrated laboratory extract at controlled doses. This is very different from drinking dandelion tea or consuming dandelion root as a food.

There is currently no strong clinical evidence showing that dandelion root extract can cure or treat colon cancer in people.

BOTTOM LINE:

DANDELION ROOT EXTRACT IS A PROMISING SUBJECT OF CANCER RESEARCH — BUT IT IS NOT A PROVEN CANCER TREATMENT.

The impressive 95% figure comes from laboratory research and should not be interpreted as a result achieved in human cancer patients.

REFERENCES:

• “Dandelion root extract affects colorectal cancer proliferation and survival through the activation of multiple death signalling pathways” — Oncotarget
• “Dandelion root extract affects colorectal cancer proliferation and survival through the activation of multiple death signalling pathways” — PubMed

DISCLAIMER:
This content is for educational and informational purposes only. It is not intended to diagnose, treat, cure, or prevent cancer or any other disease. Dandelion root should not be considered a cancer treatment or substitute for prescribed medical care. Laboratory and animal findings do not necessarily translate into benefits in humans. Never start, stop, or change cancer treatment based on information from social media. Cancer treatment decisions should always be made with qualified healthcare professionals.

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